By Gregory Hickok
Neurobiology of Language explores the learn of language, a box that has visible large development within the final twenty years. Key to this growth is the accelerating pattern towards integration of neurobiological techniques with the extra verified figuring out of language inside cognitive psychology, laptop technology, and linguistics.
This quantity serves because the definitive reference at the neurobiology of language, bringing those a variety of advances jointly right into a unmarried quantity of a hundred concise entries. The association comprises sections at the field's significant subfields, with every one part protecting either empirical info and theoretical views. "Foundational" neurobiological insurance is usually supplied, together with neuroanatomy, neurophysiology, genetics, linguistic, and psycholinguistic facts, and types.
- Foundational reference for the present kingdom of the sphere of the neurobiology of language
- Enables mind and language researchers and scholars to stay updated during this fast-moving box that crosses many disciplinary and subdisciplinary boundaries
- Provides an available access aspect for different scientists drawn to the realm, yet now not actively operating in it – e.g., speech therapists, neurologists, and cognitive psychologists
- Chapters authored by means of international leaders within the box – the broadest, such a lot specialist insurance available
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Additional info for Neurobiology of Language
2012). , 2013). After the identification of the CNTNAP2 connection, additional functional reports have further demonstrated the value of tracing FOXP2 networks for understanding language-related disorders. , 2010). (However, see Lesca et al. (2013) for evidence that casts doubt on the role of uPAR/ SRPX2 in this disorder, instead implicating a different gene, GRIN2A). Intriguingly, SRPX2 regulation by FOXP2 is thought to be an important mediator of synaptogenesis (Sia, Clem, & Huganir, 2013).
Note the emphasis on brain anatomy. 20th century (for reviews, see Damasio & Damasio, 1989; Kertesz, 1983; Shallice, 1988a). , 2003) have improved the lesion analysis method, and it continues to be a valuable method for biological inquiries in language. By their very nature, lesion analysis studies tend to relate single foci of brain injury to single psychological or linguistic phenomena. , Garrard & Hodges, 2000; Shallice, 1988b). , Plaut, 1995), this approach has been quite successful in giving insights into the neurobiology of language, that is, attributing functions to aspects of the brain (in this case, brain areas damaged by vascular lesions—or sometimes other types of lesions).
Transcription factors never act alone; they work together with other interacting proteins to regulate their targets. FOXP1 is the most similar gene in the genome to FOXP2. In some cells in the central nervous system, these two genes are coexpressed (Teramitsu, Kudo, London, Geschwind, & White, 2004), and the resulting proteins have the capacity to directly interact with each other, acting together to regulate targets in a coordinated manner (Li, Weidenfeld, & Morrisey, 2004). Rare causative mutations of FOXP1 have been implicated in a small number of cases of autism and/or intellectual disability, accompanied by notably severe speech and language problems (Bacon & Rappold, 2012).